HomeGrowth Hormone & Muscle › ACE-031

Soluble ActRIIB-Fc fusion protein / myostatin (activin receptor) decoy

ACE-031

Research use only

Written by pep4U Editorial Team · Last reviewed July 8, 2026

ACE-031 is an experimental biologic decoy receptor that blocks myostatin to promote muscle growth.

Class / typeSoluble ActRIIB-Fc fusion protein / myostatin (activin receptor) decoy
CategoryGrowth Hormone & Muscle
Status (Jul 2026)Research use only

How it works

It is a soluble form of activin receptor type IIB fused to an IgG1 Fc region; it acts as a decoy that binds myostatin (and related ligands) in circulation before they can signal muscle, removing the brake on muscle growth.

Known / used for

Developed by Acceleron and Shire and studied in clinical trials for Duchenne muscular dystrophy; development was discontinued.

Positives

Produced substantial increases in muscle mass and reported gains in bone and lean tissue in early studies.

Negatives & risks

Trials were halted in 2011 over safety concerns because the decoy also traps BMP-9 and BMP-10, causing nosebleeds, gum bleeding, and skin telangiectasia; development was formally ended in 2013.

Frequently asked questions

What is ACE-031?
ACE-031 is an experimental biologic decoy receptor that blocks myostatin to promote muscle growth. It is a soluble form of activin receptor type IIB fused to an IgG1 Fc region; it acts as a decoy that binds myostatin (and related ligands) in circulation before they can signal muscle, removing the brake on muscle growth.
What is ACE-031 used or researched for?
Developed by Acceleron and Shire and studied in clinical trials for Duchenne muscular dystrophy; development was discontinued.
Is ACE-031 FDA-approved or legal?
NOT FDA-approved. Its clinical development was halted for safety in 2011 and discontinued in 2013; it has no approved use and is prohibited by WADA under class S4.5.
What are the main risks or downsides of ACE-031?
Trials were halted in 2011 over safety concerns because the decoy also traps BMP-9 and BMP-10, causing nosebleeds, gum bleeding, and skin telangiectasia; development was formally ended in 2013.

Legal / regulatory status

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